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11.4 Metabolic and mitochondrial modulation Growing evidence indicates that metabolic inflexibility and mitochondrial dysfunction are critical contributors to ischemic injury, cardiomyocyte death, and post-infarction remodeling

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This cascade eventually generates free radicals that can initiate a wide range of toxic oxidative reactions, thereby potentially inhibiting oxidative phosphorylation and damaging mitochondrial enzymes that regulate the electron transport chain