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At 48 h, this effect was further amplified: QCN-HS achieved complete wound closure (0.0 ± 0.0 μm), significantly outperforming QCN (289.3 ± 7.17 μm) and control (482.40 ± 10.73 μm)

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The sodium-glucose co-transporter-2 inhibitor ertugliflozin modifies the signature of cardiac substrate metabolism and reduces cardiac mTOR signalling, endoplasmic reticulum stress and apoptosis